Katherine A. Bowers, PhD, MPH
- Member, Division of Biostatistics and Epidemiology
- Professor, UC Department of Pediatrics
About
Biography
My interest in research came from a long-standing desire to understand the etiology of autism spectrum disorders. After joining Cincinnati Children’s in 2012, I began to study child development more broadly. My research is primarily in perinatal epidemiology, and my specific areas of interest include exposures and conditions during pregnancy and their implications for offspring health.
Two of my primary projects focus on psychosocial factors and diabetes experienced in pregnancy. The first one is about how adversity experienced by individuals in poverty affects child development and how DNA methylation may underlie this effect. The second project follows the offspring of women who had diabetes in pregnancy. I am interested in the long-term effects of metabolic and neurobehavioral impairments.
PhD: Johns Hopkins Bloomberg School of Public Health, Baltimore, MD.
Post-doctoral Fellowship: National Institute of Child Health and Human Development, NIH, Bethesda MD.
Interests
Infant and child development; epigenetics; pregnancy; health disparities
Research Areas
Biostatistics and Epidemiology
Publications
Association between maternal prenatal depressive symptoms and offspring epigenetic aging at 3-5 weeks. Annals of Epidemiology. 2024; 93:1-6.
Current Recommended Estrogen Dosing for Pubertal Induction in Turner Syndrome Results in Normal Uterine Growth. The Journal of Clinical Endocrinology and Metabolism. 2024; 109:e1040-e1047.
Neonatal AVPR1a Methylation and In-Utero Exposure to Maternal Smoking. Toxics. 2023; 11.
THU203 Self-puberty Staging In Endocrine Encounters During The COVID Pandemic. Journal of the Endocrine Society. 2023; 7:bvad114.1454.
1149-P: Health Literacy and Numeracy Based on The Newest Vital Sign Is Not Associated with Hemoglobin A1c in Caretakers or Youth with Type 1 Diabetes. Diabetes. 2023; 72.
Epigenome-wide association of neonatal methylation and trimester-specific prenatal PM2.5 exposure. Environmental Epidemiology. 2022; 6:e227.
Epigenome-Wide Association of Neonatal Methylation and Prenatal PM2.5 Exposure Suggests Timing-Specific Effects. ISEE Conference Abstracts. 2022; 2022.
Association Between Maternal Adverse Childhood Experiences and Neonatal SCG5 DNA Methylation-Effect Modification by Prenatal Home Visiting. American Journal of Epidemiology. 2022; 191:636-645.
Caregivers of individuals with Rubinstein-Taybi syndrome: Perspectives, experiences, and relationships with medical professionals. Journal of Genetic Counseling. 2022; 31:153-163.
Substance Use and Utilization of Prenatal and Postpartum Care. Journal of Addiction Medicine. 2022; 16:84-92.
From the Blog
DNA Methylation May Serve as Biomarker for Maternal Stress Passed to Children
Katherine A. Bowers, PhD, MPH, Alonzo T. Folger, PhD, MS3/21/2022
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